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As a service to the Bay Area community, FORE is proud to offer a wonderful group of osteoporosis education speakers... more
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Mechanisms, Evaluation and Treatment of Systemic & Focal Bone Loss in Rheumatic Disease
Webinar Professor Series
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One AMA PRA Category 1 credit is offered per series, credit may also be applied to the CMA Certification in Continuing Medical Education. Physicians should only claim credit commensurate with the extent of their
participation in the activity.
At the end of the sessions, participants will be able to:
Series #1: Cytokine Networks in Systemic & Focal Bone Loss
- Illustrate how cytokines participate in physiological bone remodeling
- Describe the mechanism of increased rate of bone remodeling in pathological states
- Discuss the cellular mechanisms and important molecular events that lead to bone erosions
Series #2: Can Medications Inhibit & Even Heal Erosions in RA?
- Describe radiographic scoring methods for progression of RA
- Recognize that current medications can inhibit progression of erosions
- Discuss the evidence that medication reverse existing erosions in RA
Series #3: Epidemiology of Bone Loss in Inflammatory Arthritis and Secondary to Glucocortocoids
- Evaluate the magnitude of bone loss and fractures in patients with inflammatory arthritis
- Describe the etiology of glucocorticoid induced osteoporosis
- Discuss the magnitude of glucocorticoid-induced bone loss in inflammatory arthritis
Series #4: New Paradigm in Osteoporosis Assessment: Absolute Fracture Risk Determination
- Integrate clinical risk factors in determining fracture risk
- Analyze the concepts of international reference standards, fracture risk assessment and treatment thresholds
- Discuss the future WHO recommendations and their implications for clinicians
Series #5: Novel Therapies Directed at Molecular Targets for Bone Loss
- Understand the novel molecular targets to treat systemic and focal bone loss
- Describe how manipulating molecular targets can define future osteoporosis treatment
- Discuss the treatment of focal and systemic bone loss with agents directed at RANK-L, Cathepsin K, aVß3 integrins, etc.